Upcoming Talks
I have two talks planned over the next two months. Hope to see you there!
Ancestral Health Symposium 2014: UC Berkeley, August 7-9
If you want to understand the most rigorous science available on leptin resistance-- a key mechanism of obesity and a major barrier to fat loss-- this talk is for you. This is my primary area of professional expertise; I have years of firsthand research experience on the subject and I've published a number of related papers in peer-reviewed journals. The talk will be accessible to nearly all levels of expertise. AHS14 tickets are available here. I've pasted the talk's abstract below.
What Causes Leptin Resistance?
Leptin is the primary hormonal regulator of body fatness. Obese people exhibit a resistance to leptin�s effects in the brain, causing the brain to oppose fat loss by multiple mechanisms. Research in animal models suggests that leptin resistance may be required for obesity to develop. How does leptin resistance occur, and what causes it? Research has not yet provided us with definitive answers, but several plausible possibilities have emerged. This talk will review what is known about leptin resistance and its causes.
McDougall Advanced Study Weekend: Santa Rosa, CA, September 5-7
Dr. John McDougall invited me to speak at his yearly symposium after viewing my TEDx talk "The American Diet: a Historical Perspective". I look forward to sharing my thoughts and interacting with a different audience than I'm used to. The talk will be an expanded version of the one I presented at AHS13. Tickets are available here. I've pasted a modified version of my AHS13 abstract below.
Insulin and Obesity: Reconciling Conflicting Evidence
The pancreatic hormone insulin regulates the trafficking and metabolism of carbohydrate and fat, and its secretion is particularly stimulated by carbohydrate and protein. Since circulating insulin is elevated in common obesity, and insulin influences fatty acid flux into and out of fat tissue, this has raised the possibility that elevated insulin causes common obesity, and that dietary carbohydrate is particularly fattening. A large amount of evidence appears to support the hypothesis that insulin causes obesity, and a large amount of evidence appears to falsify it. This presentation will outline a framework capable of reconciling this seemingly conflicting evidence.
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Ancestral Health Symposium 2014: UC Berkeley, August 7-9
If you want to understand the most rigorous science available on leptin resistance-- a key mechanism of obesity and a major barrier to fat loss-- this talk is for you. This is my primary area of professional expertise; I have years of firsthand research experience on the subject and I've published a number of related papers in peer-reviewed journals. The talk will be accessible to nearly all levels of expertise. AHS14 tickets are available here. I've pasted the talk's abstract below.
What Causes Leptin Resistance?
Leptin is the primary hormonal regulator of body fatness. Obese people exhibit a resistance to leptin�s effects in the brain, causing the brain to oppose fat loss by multiple mechanisms. Research in animal models suggests that leptin resistance may be required for obesity to develop. How does leptin resistance occur, and what causes it? Research has not yet provided us with definitive answers, but several plausible possibilities have emerged. This talk will review what is known about leptin resistance and its causes.
McDougall Advanced Study Weekend: Santa Rosa, CA, September 5-7
Dr. John McDougall invited me to speak at his yearly symposium after viewing my TEDx talk "The American Diet: a Historical Perspective". I look forward to sharing my thoughts and interacting with a different audience than I'm used to. The talk will be an expanded version of the one I presented at AHS13. Tickets are available here. I've pasted a modified version of my AHS13 abstract below.
Insulin and Obesity: Reconciling Conflicting Evidence
The pancreatic hormone insulin regulates the trafficking and metabolism of carbohydrate and fat, and its secretion is particularly stimulated by carbohydrate and protein. Since circulating insulin is elevated in common obesity, and insulin influences fatty acid flux into and out of fat tissue, this has raised the possibility that elevated insulin causes common obesity, and that dietary carbohydrate is particularly fattening. A large amount of evidence appears to support the hypothesis that insulin causes obesity, and a large amount of evidence appears to falsify it. This presentation will outline a framework capable of reconciling this seemingly conflicting evidence.